Posting Product About Finiti From Jeunesse Globa Distributor USA,
Jeunesse's FINITI™ Enhances Telomere Length
According to Wikipedia, a telomere is a region of repetitive nucleotide sequences at each end of a chromatid, which protects the end of the chromosome from deterioration or from fusion with neighboring chromosomes.
Watch this video and find out the connection between short telomeres and ageing, and how Jeunesse's FINITI™ can effectively enhance telomere length, effectively halt, and reverse aging.
FINITI™ is one of the latest products release by Jeunesse, the only networking company that has truly embraced the concept of youthful aging. This product supports the maintenance of your natural DNA repair mechanisms. FINITI™ is based on a Nobel Prize winning research. This the answer to the challenge of aging. See More
About Finiti Face on Video Youtube :
Announcing a revolutionary development in anti-aging and science called FINITI™: a dietary supplement based on Nobel Prize winning research.
To appreciate the importance of this groundbreaking product, the causes of aging must be understood at a cellular level. The problem begins in a compound called a telomere. Telomeres are made of repetitive DNA sequences that protect the ends of human chromosomes. At birth, telomeres are long. As time goes on, telomeres begin to shorten and fray like the ends of an old shoelace. When telomeres become too short, cells can no longer carry out their critical functions. A process then begins called apoptosis–in which cells start to self-destruct and die. As cells stop functioning, they can spew out dangerous free radicals and inflammatory molecules that can damage nearby cells. There begins the journey into what is known as cellular aging.
Thousands of studies show the connection between short telomeres and the process of cellular aging. A newly patented nutrient known as TA-65 MD, has been shown to add length to short telomeres by transiently activating the enzyme called telomerase. Moreover, it is the only known substance in history capable of doing this.
TA- 65 MD is the key component in FINITI™; it is also the only nutrient existing that can lengthen short telomeres. Accompanying TA-65 MD is a list of powerful ingredients. COQ-10 is one of them; a vital antioxidant that has the ability to protect against free radicals, it reduces the oxidative stress that leads to cellular damage. Fucoidan and purslane extract support adult stem cells, which are important in cellular rejuvenation and renewal, and help to maintain a healthy immune system. Vitamins C, B6, B12, and Folate are also included to encourage critical metabolic functions. These list only a few of the ingredients and benefits FINITI™ offers.
Jeunesse® first announced the release of FINITI™ in February, 2013. FINITI™ reinforces the maintenance of natural DNA repair mechanisms, promotes a feeling of youthful vitality, and supports the body's natural rejuvenation system.
About Jeunesse Global®
Jeunesse® is a leading network marketing company based in Orlando, FL that specializes in providing youth-enhancing solutions based on some of the latest science available. Their research focus on adult stem cell technology, telomere support, DNA repair, and nutrigenomics. Products are made in the U.S.A. and are exclusively formulated for Jeunesse®. With a multi-lingual customer service, back office support team, global enrollment system, and in-house programming already in place, the company is fully operational in 20 offices around the world. Its shipping paths extend to over 85 countries. For more information, please visit www.jeunesseglobal.com. Jeunesse® and the Jeunesse® logo are registered trademarks of Jeunesse Global®, LLC in the U.S. and/or other countries.
We all understand getting older. That is simply the passage of time. But along with that comes diminished physical and mental ability and all the signs of what we call aging. You know, things like wrinkles, loss of hair, less flexibility and strength, greater susceptibility to illness and disease. In fact many diseases are age-related; they are more likely to occur in one’s later years. These include heart disease, arthritis, strokes, poor short term memory, Alzheimer’s, and so on.
The debilitating effects of aging are major reasons that humans have long searched for the elusive “fountain of youth”. If we could just stave off the aging process we might achieve immortality, or at least live well beyond the norms for our times. While we have not achieved that immortality, in the last 200 years or so, the average life span in the Americas has risen from about 25 years in 1700′s to at or above 80 years in the 21st Century.
Advances in science, pharmacology and medicine have contributed much to this greater life expectancy. We live better, eat better and have all the advantages of modern civilization compared to our 18th Century progenitors. We are still subject to the laws of nature and the various factors that cause us to show our age. There are a number of theories about this aging process, none of which is likely to be the single most important; a combination of factors acting in concert is most probable. So let’s take a quick look at some of these theories.
1. Wear and Tear Theory
As young organisms we move with smooth grace and fluidity because our joints, tendons and muscles are supple yet strong. All systems operate at top efficiency. But eventually our joints stiffen, muscles shrink, cartilage becomes worn and we really slow down with age. This is wear and tear over the years on all systems, not just the joints and muscles. The processed foods we eat, pollutants and carcinogens in the air we breathe and water we drink, and the stresses of infective agents just wear us down over time to the point where we can no longer repair the damages. This is the biogrind as Dr. White would say. As time goes on the damage done to our cells, tissues and organs accumulates because we can’t repair and regenerate quickly enough, and we simply wear out.
2. Waste Accumulation Theory
Over time our cells are thought to accumulate waste products because they cannot be disposed of quickly enough. When we are young our bodies and systems are growing quickly because anabolic metabolism (construction in effect) outstrips catabolic metabolism (destruction). Wastes are natural byproducts of these metabolic processes. In young, vigorous cells waste material is easily broken down and excreted, but as the cells get older they are less efficient and wastes accumulate. This leads to impaired function and eventually cell death which eventually results in the overall effects we call aging.
A visible example of this is the accumulation of pigment called lipofuscin in skin cells, resulting in what are known as age spots. Lipofuscin builds up in the cells of the skin, nervous system and heart, for instance, and leads to deterioration of brain and heart function over time.
3. Free Radical Theory
Way back in 1954 Dr. Denham Harman suggested that aging is caused by free radical reactions that may be caused by the environment, disease and as part of the normal aging process. Fifteen years later it was recognized that free radicals were actually also produced within the body as a result of normal metabolic activities within our cells. At the same time it was recognized that some free radicals (superoxide and hydroxyl radicals) were causative factors in some degenerative diseases and in aging.
Free Radical EffectsFree radicals are atoms or groups of atoms that have an imbalance of electrons and are therefore chemically unstable and very reactive. A free radical needs another charged atom or group of them to bond with in order to become stable. As they move throughout the body, these free radicals combine with atoms in the various cells and tissues in order to neutralize their electrical charges and become stable. This process causes damage to the target cells and tissues.
The effect can be likened to the rusting of metal when it comes into contact with water and oxygen. In the case of our cells and tissues, the effect is similar to the oxidation (rusting) of metal except that it occurs within our bodies. It is in effect biological rusting. Many free radicals do contain highly unstable oxygen atoms and are thus called Reactive Oxygen Species (ROS). Free radicals attack tissues and cells by tearing away atoms from the cell structures, damaging them directly and often producing new free radicals in the process.
Free radicals attack cells and tissues throughout the body including structures within the cells such as DNA, RNA, chromosomes and mitochondria. There are numerous types and classes of free radicals and their effects are extremely varied. This is a complex topic which will be covered more in depth in future posts.
Our cells do produce natural products called antioxidants which can neutralize specific free radicals. This is a topic for future posts as well.
4. Calorie Restriction Theory
This theory suggests that a diet that provides optimal nutrition with a minimum of calories will promote longevity, or in other words, retard the effects of aging. This suggests that high caloric intake can accelerate the effects of aging. It is true that individuals who are overweight or obese tend to have more health problems such as cardiovascular disease and diabetes. Such individuals are thought to have a lower life expectancy than fitter individuals.
Low calorie diets have become very popular in some regions as a result of the implications the caloric restriction theory has to the aging process and the potential for extending ones life span.
5. Thymic Stimulating Theory
Thymus GlandThe thymus gland has been called the gland of youth because it is large (200-250 grams) at birth but by the time you reach the age of 60, it has shrunk to a tiny fraction of that. Relatively recent research has shown that the thymus is very important in the development and activity of our immune system. For instance, white blood cells programmed in the thymus (T-lymphocytes) are known to be very important as killers of viruses and cancer cells. A strong immune system will definitely reduce the aging effects of pathogens and disease, thus theoretically playing a significant role in the aging process.
6. Mitochondrial Theory
Mitochondria are minute organelles within our cells. They are the power plants responsible for release of energy from nutrients taken into the cells Mitochondrionthrough the process we know as oxidative phosphorylation. The chemical processes within the mitochondria that produce the energy needed for all bodily functions also generates free radicals. Mitochondria are very susceptible to damage from free radicals, including the DNA within the mitochondria. Damage from free radicals accumulates because mitochondria cannot be repaired or replaced. So as we age, mitochondrial function declines and mitochondria are lost. Consequently, cells and tissues lose functionality, we are more easily fatigued, we become more susceptible to disease and we suffer many other effects of aging.
Interestingly, the DNA in your mitochondria is derived from your mother (maternal DNA). Scientists have used this fact to trace the lineage of various races of people around the globe back to common female ancestors. Your common paternal ancestor is left shrouded in mystery!
7. Hayflick Limit Theory
This theory was postulated in 1961 by two cell Biologists, Hayflick and Moorehead, hence the name. Their experiments suggested that the aging process of cells was governed by a biological clock within each cell that limited it to a finite number of cell divisions. Once the limit was reached they suggested that the cell ceased to divide (or replace itself) and would eventually die. In their system the limit on the number of divisions was about 50, but these divisions were spread over a variable length of time depending on the nutrient status of the medium in which they were grown.
Cells with an over abundance of nutrients went through the process three times as fast as those grown in normal nutrient conditions. Cells grown in nutrient restricted media took three times as long as normal to go through the full cycle. On the other hand, some cells never reached the 50 divisions limit because of waste accumulation and damage to cell membranes, mitochondria, DNA and so on. All this suggests the degeneration of cells and loss of cells (before or after the divisions limit) could explain some of the effects of aging. It also includes elements of some of the other theories described above.
8. The Telomerase Theory
TelomeresThis to me is one of the more interesting theories of aging and is an area of ongoing research. Telomeres are small sections of DNA that extend from the tips of chromosomes. As the cell divides, the chromosomes also divide so that a full set of chromosomes can be passed to the new cells. At each division of the chromosome, the telomeres get shorter. After a certain number of divisions the telomeres are reduced to a very small fraction of their original size at which time the cell ceases to divide and eventually dies. This in fact may be the internal clock that was postulated in the Hayflick Theory.
Here’s the most interesting part to me anyway. The length of the telomere is controlled by an enzyme called telomerase from whence the name of the theory comes. In young cells, the level of telomerase is high which maintains the length of the telomeres as cell divisions occur. As the cells age, telomerase levels decline and so does the length of telomeres. Research has shown that if telomerase levels are kept high (in experimental systems), cells continue to divide indefinitely.
Telomerase production is controlled by a specific gene. That gene is thought to be turned on by Human Growth Hormone (HGH). If so, the combination of HGH and Telomerase could have fundamental effects on the aging process.
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